Alzheimer’s Breakthrough with Dr. Josh Helman, MD
Oct 22, 2026 · 1 hr 18 min · 12 segments
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Josh HelmanHost
And just so people understand, the mold spores, the live mold, things like black mold, aspergillus, they can be toxic to people just from the allergic, the live mold itself, right? And that's number one.

But number two, many of these molds will produce toxic, will produce fatty toxins that can then go into people, into our fat cells, our fatty brain and other fatty tissue and lie there.

waiting until the fat toxins get redistributed, and they're both two different problems from the same organism, and they have to be treated differently.

and it's been studied because these mold toxins can very quickly damage rapidly dividing cells in the gut and elsewhere in the body and yes unfortunately it has been used and studied as a biological weapon we want to help people my patients and others who have already been exposed to these fatty mold toxins.

This is not something I learned in my traditional training in emergency medicine or in lifestyle medicine or in medical school.

But having for over 10 years treated patients with mold injury, even before we had the spike proteins come into population, I've been humbled and I appreciate how devastating it can be to people and their immune system and their other systems.
When we start discussion about the study that we would show where the LPS, which is the bacterial toxins and then mold and mold related toxins or mold produced toxins, fatty toxins, as Dr. Josh said, and the spike proteins presence and the convergence of those, I want to give some clues to our audience to help understand the discussion.
So on the surface, it may look like a calm cell afterwards, but it has changes that have become established.
And the next time any other similar event happens, for example, inflammatory event, will cause that cell to respond in a dysregulated fashion.
So it may respond faster, it may respond more exaggerated, or it may respond by dying or by doing incorrect things.
And the example for that that comes to me is that imagine if I am standing on a roadside and somebody just approaches me and I'm thinking they're friendly and they come in and they punch me and they move on.
That would now, let's say I recover from that punch and I'm looking normal again.
But I have now inside of me a training that, hey, somebody could come in and punch me.

And just so people understand, the mold spores, the live mold, things like black mold, aspergillus, they can be toxic to people just from the allergic, the live mold itself, right? And that's number one.

But number two, many of these molds will produce toxic, will produce fatty toxins that can then go into people, into our fat cells, our fatty brain and other fatty tissue and lie there.

waiting until the fat toxins get redistributed, and they're both two different problems from the same organism, and they have to be treated differently.

and it's been studied because these mold toxins can very quickly damage rapidly dividing cells in the gut and elsewhere in the body and yes unfortunately it has been used and studied as a biological weapon we want to help people my patients and others who have already been exposed to these fatty mold toxins.

This is not something I learned in my traditional training in emergency medicine or in lifestyle medicine or in medical school.

But having for over 10 years treated patients with mold injury, even before we had the spike proteins come into population, I've been humbled and I appreciate how devastating it can be to people and their immune system and their other systems.
When we start discussion about the study that we would show where the LPS, which is the bacterial toxins and then mold and mold related toxins or mold produced toxins, fatty toxins, as Dr. Josh said, and the spike proteins presence and the convergence of those, I want to give some clues to our audience to help understand the discussion.
So on the surface, it may look like a calm cell afterwards, but it has changes that have become established.
And the next time any other similar event happens, for example, inflammatory event, will cause that cell to respond in a dysregulated fashion.
So it may respond faster, it may respond more exaggerated, or it may respond by dying or by doing incorrect things.
And the example for that that comes to me is that imagine if I am standing on a roadside and somebody just approaches me and I'm thinking they're friendly and they come in and they punch me and they move on.
That would now, let's say I recover from that punch and I'm looking normal again.
But I have now inside of me a training that, hey, somebody could come in and punch me.
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