Severe acute respiratory syndrome coronavirus 2

The Tiger of Vietnam or something like that.

So he comes from good stock, and he conducted a trial where he took what they considered to be purified SARS-CoV-2, took healthy people, put them up their nose, and they did nothing.

So if they are making a bioweapon, it's not very good because it doesn't do anything to people.

You know, a couple of people had a scratchy nose.

They are told to, in their protocol... as a top-down measure, they get told to starve these cells and produce the observable effect that they blame on viruses that is fraudulent.

And so we took that, we put it under a microscope, and we looked in the Petri dish, and we saw 27 different types of viruses in in a cell line which couldn't have possibly have contained any viruses.

We saw things that looked identical to Ebola, to smallpox, to chickenpox, to HIV, to SARS-CoV-2, 27 different morphologies of these cell lines.

And what's happening is that you get a cell, it's a very, very basic thing, all of cell biology that they've taught in the 1800s and 1900s that was propagated from the Royal Society as bollocks.

Before diving into it, I was kind of of the mindset, it was like probably 60-40 in favor of the lab leak versus the zoonotic origin thing.

And then when I started digging into a lot of the science behind it, my opinion shifted heavily in the direction of zoonosis such that I feel like it's almost inconceivable that it comes from a lab at this point, if you've been following any of the science related to the origin of the SARS-CoV-2 virus.

So I guess my story that I'm going to tell is going to be basically the bog standard establishment story of there was some intermediary animal, a pangolin or something, at the wet market.

A vendor there got infected.

We know how COVID spreads.

So if somebody starts off getting sick at the wet market, it spreads in exactly the way we would expect it to spread.

I know that there are intermediary animals that could host a SARS-CoV-2 virus.

Pangolin was, I think, the most likely identified candidate.

We've got Jim Haslam with us, author of "COVID-19: Mystery Solved: It Leaked From Wuhan Lab But It's Not Chinese Junk." His, uh, x.com is jhas5.

His Substack is jimhaslam.substack.com, where he talks about the reverse engineering of origins of SARS-CoV-2.

And George Howard is with us as well, the pajama scientist himself, from x.com/cosmiktusk and carolinafloo.org.

So welcome-

In fact, he's so well-revered that the US government turned to Ralph Baric, this professor at the University of North Carolina, and they asked him point-blank very early in the pandemic.

You can read the email in The New York Times.

It's in, called "The Red Dawn." And the bureaucrats, the bio bureaucrats, you know, uh, Department of Health, you know, DoD, uh, biodefense kind of guys, asked him point-blank, "Ralph Baric, is this thing engineered? Is SARS-CoV-2 engineered?" And he replied back very quickly.

He usually doesn't reply to emails 'cause he gets so many of them.

It copies that DNA that we want to look for.

And I think when PCR came along and, of course, when SARS-CoV-2 started up and we had this global pandemic, it became clear that you could use a monitoring of untreated wastewater, go upstream and see what was happening in the community.

And, you know, so it was really a paradigm shift for many of us that worked in wastewater to think about going upstream.

What's happening in the community? Because we, you know, we could find all these pathogens in wastewater, but we never mathematically or statistically in time and space tried to connect what was in sewage to what was happening in the community.

We haven't done surveillance yet, only in wastewater systems from Michigan.

Okay.

And because we have been monitoring the wastewater since SARS-CoV-2, and we've saved some of our samples, we're going to go back in time around Michigan now.

Yeah.

It's hard not to, uh, believe that.

Without dispute, the early news and images coming out of China, Italy, and New York City, as they were coping with SARS-CoV-2, were alarming.

Images of responders and health officials in moon suits, and reports of COVID hotspots and deaths dominated news cycles.

We didn't know if this new virus would be as deadly as Ebola, with an infection fatality rate of 40%, MERS at 30%, or S- or C- SARS-CoV-1 at an eight to, to 10% infection fatality rate.

But by March 17th, 2020, in an article published in Stat News, Professor, Professor-- Stanford Professor John Ioannidis analyzed the data from the Diamond Princess cruise ship that had been quarantined due to a COVID outbreak.

From that data, Professor Ioannidis, Ioannidis projected an infection fatality rate for SARS-CoV-2 of 0.125%, 0.125%.

The predicted range is somewhere between 0.05 to 1%.

Now, per- perspective, an average flu season has an infection fatality rate of approximately 0.1%.

W- with the process one, they were basically using a PCR process to duplicate DNA, right? That's what they do with, with PCR.

They wanna check do you have any of, any, any of this, uh, tr- uh, remnant of DNA in your nose that we can say was, you know, COVID, was SARS-CoV-2 or whatever, right? So they replicate it, right? That's the whole idea behind the PCR.

And it's relatively clean to do it that way, and they were also using a very, very high quality, uh, s- uh, system using these, um, magnetic, these sort of magnetic beads to take out any impurities after that PCR process was used, again, to create the template where then they, you know, they, they use that to generate the MR- the, the mRNA.

In process two they use a different method to generate the MR- the DNA backbone, okay? Where they basically have a DNA template that they put into this thing called a plasmid, um, which is a part of a bacteria.