Jun 5, 2026 · 1 hr 26 min · 12 segments
Jack Schroder is a Certified Health Coach and Nutritionist who specializes in helping people restore their health through a holistic, root-cause approach. His passion for health coaching was born from…
Jack SchroderGuestRyan Mitchell BrownHostChrisHost
Um, so yeah, I suppose with the mitochondria, uh, yeah, I, I think you've, you've already summarized it quite beautifully here around its interactions in terms of picking up the slack from other ones, communicating on like not just like a mitochondrial to mitochondrial level, but cell to cell level, tissue to tissue level.

I think there's a lot of that stuff going on, on like the quantum scale in terms of communication of, you know, shuttling energy and information throughout the system, and that's gonna look like, um, through water networks and microtubules and through melanin and other semiconductors and fascia and everything else in between.

And I suppose like with researching that, there's a lot of amazing like hypotheses around it, and there's also like a lot of unknowns.

So firstly, I suppose like the, the biggest consideration is like there's all these pathways that obviously lead into the mitochondria.

We've got the TCA cycle, we've got anaplerosis, cataplerosis, we've got amino acids feeding in, we've got fatty acids coming in, we've got glucose coming in.

And now I would say like the, the biggest thing that is going to dictate what is going to go on within our cells, within our tissues is ultimately how oxygen gets reduced at, uh, cytochrome C oxidase.

So that would be the make or break in terms of obviously everything that sort of builds up to that.

And what essentially happens at that step will govern life as we know it because ultimately that's where oxygen is getting reduced with the electrons be- being fed through.

And as we know, like oxygen is the most electronegative, uh, or second most electro- most second most or most electro- electronegative.

So electrons flow through and it gets reduced to water, and that water is obviously very important within itself.

It is deuterium depleted and it plays a very fundamental role within the mitochondria and arguably throughout the cell.

So I would say like-Anything that compromises that step, whether it's like a nutrient deficiency, for example, maybe it's coenzyme Q10, maybe it's riboflavin, maybe it is, um, vitamin B3, m-maybe it's like a antioxidant-based molecule, maybe it's glutathione or thioredoxin or something like that.

Anything that essentially compromises the reduction of oxygen is going to govern the, the pulses of free radicals and the biophoton emissions that come with it, and also the, again, the energy versus exhaust, where energy reflects...

Well, on the biochemical level, it reflects ATP, and then the exhaust refle- will reflect, uh, reactive oxygen and nitrogen species, which then again, like when they, uh, get neutralized and reduced, they will shoot out certain, uh, wavelengths of biophoton emissions.

Um, so yeah, I suppose with the mitochondria, uh, yeah, I, I think you've, you've already summarized it quite beautifully here around its interactions in terms of picking up the slack from other ones, communicating on like not just like a mitochondrial to mitochondrial level, but cell to cell level, tissue to tissue level.

I think there's a lot of that stuff going on, on like the quantum scale in terms of communication of, you know, shuttling energy and information throughout the system, and that's gonna look like, um, through water networks and microtubules and through melanin and other semiconductors and fascia and everything else in between.

And I suppose like with researching that, there's a lot of amazing like hypotheses around it, and there's also like a lot of unknowns.

So firstly, I suppose like the, the biggest consideration is like there's all these pathways that obviously lead into the mitochondria.

We've got the TCA cycle, we've got anaplerosis, cataplerosis, we've got amino acids feeding in, we've got fatty acids coming in, we've got glucose coming in.

And now I would say like the, the biggest thing that is going to dictate what is going to go on within our cells, within our tissues is ultimately how oxygen gets reduced at, uh, cytochrome C oxidase.

So that would be the make or break in terms of obviously everything that sort of builds up to that.

And what essentially happens at that step will govern life as we know it because ultimately that's where oxygen is getting reduced with the electrons be- being fed through.

And as we know, like oxygen is the most electronegative, uh, or second most electro- most second most or most electro- electronegative.

So electrons flow through and it gets reduced to water, and that water is obviously very important within itself.

It is deuterium depleted and it plays a very fundamental role within the mitochondria and arguably throughout the cell.

So I would say like-Anything that compromises that step, whether it's like a nutrient deficiency, for example, maybe it's coenzyme Q10, maybe it's riboflavin, maybe it is, um, vitamin B3, m-maybe it's like a antioxidant-based molecule, maybe it's glutathione or thioredoxin or something like that.

Anything that essentially compromises the reduction of oxygen is going to govern the, the pulses of free radicals and the biophoton emissions that come with it, and also the, again, the energy versus exhaust, where energy reflects...

Well, on the biochemical level, it reflects ATP, and then the exhaust refle- will reflect, uh, reactive oxygen and nitrogen species, which then again, like when they, uh, get neutralized and reduced, they will shoot out certain, uh, wavelengths of biophoton emissions.
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