Molecular: Steroid Tier List (Science-Based Version) + Ways To Reduce Damage

75 milligrams of mint.

And he was on 80 milligrams, 90 milligrams of Aromasin a week just to keep his estrogen in range, so his blood pressure wasn't really high.

It's just ridiculous to have to do things like that.

It seems to cross the blood-brain barrier really well.

It's part of the reason that it's poorly SHBG bound as well.

There's actually a study on it where the concentration in the brain is a lot higher than testosterone's concentration.

Testosterone's like three to one.

With menth it's literally one to one.

So you're getting a large amount just floating around in your brain, which is never a good thing.

It just causes problems with neuroinflammation, calcium channel overload, glutamate signaling.

These 19-nors, they shut that off, so you have no break, and then you have increased sympathetic tone on top of it, and that kind of creates a situation where you have these neurons that basically just kind of commit suicide, essentially.

It's not a good idea because you're gonna deplete your iron stores over time.

Like, I can't tell you how many people I see that over time, from giving blood so frequently, like every three or four months, they'll just have iron ferritin that's basically, like, 20 or 15.

They're literally clinically anemic.

What you can do, nattokinase, telmisartan, or any angiotensin receptor blocker to bring down the hematocrit.

If things are really critical, you can use pentoxifylline to increase your red blood cell deformability and potentially reduce some kidney-related inflammation, which will make your blood flow better.

But that is sort of a Band-Aid on top.

So fenofibric is one of those things that can sort of reduce cardiac hypertrophy over time, especially if you're running higher doses.

But you do have to keep in mind it will raise your potassium, and hyperkalemia will put you in the hospital.

75 milligrams of mint.

And he was on 80 milligrams, 90 milligrams of Aromasin a week just to keep his estrogen in range, so his blood pressure wasn't really high.

It's just ridiculous to have to do things like that.

It seems to cross the blood-brain barrier really well.

It's part of the reason that it's poorly SHBG bound as well.

There's actually a study on it where the concentration in the brain is a lot higher than testosterone's concentration.

Testosterone's like three to one.

With menth it's literally one to one.

So you're getting a large amount just floating around in your brain, which is never a good thing.

It just causes problems with neuroinflammation, calcium channel overload, glutamate signaling.

These 19-nors, they shut that off, so you have no break, and then you have increased sympathetic tone on top of it, and that kind of creates a situation where you have these neurons that basically just kind of commit suicide, essentially.

It's not a good idea because you're gonna deplete your iron stores over time.

Like, I can't tell you how many people I see that over time, from giving blood so frequently, like every three or four months, they'll just have iron ferritin that's basically, like, 20 or 15.

They're literally clinically anemic.

What you can do, nattokinase, telmisartan, or any angiotensin receptor blocker to bring down the hematocrit.

If things are really critical, you can use pentoxifylline to increase your red blood cell deformability and potentially reduce some kidney-related inflammation, which will make your blood flow better.

But that is sort of a Band-Aid on top.

So fenofibric is one of those things that can sort of reduce cardiac hypertrophy over time, especially if you're running higher doses.

But you do have to keep in mind it will raise your potassium, and hyperkalemia will put you in the hospital.