Yeah

... no matter what outcome you get.

And so you have to have a study big enough to put into play all the variables, the likely responsiveness.

And so you, uh, I end up having a study which is powered to give you, if you have a certain number of people, uh, expecting a certain percenti- You're aiming at saying, "Look, I'm expecting a 30%, um, preferential outcome for this drug for, for this particular disease.

And to get a 30% outcome, I need to power it by having 200 people or 300 people-

Give us, I'll give you an example, yeah

...

I'll give you an ex- give you an example.

Yeah

... no matter what outcome you get.

And so you have to have a study big enough To put into play all the variables, the likely responsiveness.

And so you, uh, I end up having a study which is powered to give you, if you have a certain number of people, uh, expecting a certain percent.

You're aiming at saying, "Look, I'm expecting a thirty percent, um, preferential outcome for this drug for, for this particular disease." And to get a thirty percent outcome, I need to power it by having two hundred people or three hundred people-

Yeah.

Give you an ex- give you an example.

Um, the, there's been a number of studies showing that if you have a active flu vaccine, that that flu vaccine, uh, significantly reduces your chance of getting serious, uh, infection from COVID for a short period of time.

Mm

... that people, they still believe it.

I, I had a, a very good friend who I, uh, was in my group i-in medicine wh-when we trained, um, ring me last night, uh, to say that, uh, one of his...

He's a very good, experienced, uh, general practitioner, and he said that, uh, one of his patients, uh, contacted him th- yesterday to say could he come and have his seventh, seventh vaccine.

And, uh, I, I'd run into, to Jo- also John, uh, at a medical meeting the other night, and we were chatting about various things, and he said, "Oh, I didn't realize you had an interest in COVID." He said, "I, I was one of the pastors, 'cause I'm ready, I'm ready for my seventh, uh, vaccine." And so I, I had a little chat with him, and, um, he said he had no idea, no idea that drugs like ivermectin actually had evidence that they worked.

Yeah

YeahSo they, they did, uh, a study in, of, uh, using this messenger RNA, uh, and it was ju-just basically at the same time as the Premier of Victoria saying she...

Uh, and what she said was that, "We're gonna make this.

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That virus, the spike protein, then you're not essentially immunizing in a totally naive way.

And so The response you get is not just a good positive response, although initially you will get a positive response, but as experience goes and as you get more exposure to the virus in the environment and as you get more of these booster vaccines, particularly after about three of them in a year or two, you actually get a net turn off rather than a net turn on.

So instead of getting terrifically vigorous immune response, which initially when the vaccine rollout occurred, there was a very good protective immune response.

But progressively, that immune response has become less and less, which means that there's less and less protection from the vaccine.

And the dynamic that needs to be understood, John, is a dynamic that involves three components.

The first component, is the virus itself.

That was effective at preventing hospitalizations.

Well, there's been a lot of talk around the

topic of negative immunity.

And I think people mustn't be surprised.

There's all sorts of comments that would suggest, oh dear me, we're getting more infections, not less.

Vaccines are supposed to prevent, not promote infection.

You need to understand the reasons for this.

The COVID virus is an inhaled virus.

we do that, Robert, why was it that the vaccine in the early days in Australia did seem to be effective, in fact, was effective at preventing hospitalisations?

What sort of things would concern you about the mRNA vaccines that would indicate caution?

Well, I look at it very, very simply.

The first is that messenger RNA vaccines potentially, as we talked earlier, can infect every cell in the body.

And when it does that, it can create every cell into potentially a target for destructive T cells to damage it.

And that means that not just traditional autoimmune disease, but a whole range of diseases we haven't seen or examples of which we've only seen rarely might occur.

It means that there can be long-term post-COVID vaccine as well as post-COVID long COVID.

I see far more post-COVID vaccine long COVID in my clinic than I do post-COVID long COVID, which is the one everyone knows about.

Consulta a tu médico si los síntomas persisten.

Remind me, what was happening? It was causing severe

disease, wasn't it? 12.5% of babies, little infants, who were given the active got life-threatening RSV infection because every kid gets RSV, not once, twice, they'll get it a dozen times.

I mean, it can be quite nasty, it's not good, but it's got a very peculiar type of relationship with the body's immune system and we've known since the 1960s that you don't fiddle with RSV.

because back then, with antigen vaccines, they were getting kids dying from overwhelming RSV infection.

So it's going to be a lot worse, in my view, by putting in an messenger RNA vaccine where you can't even control the dose or the nature of the antigen that's expressed, and that's also probably important.

are back? Remember to grab a few extra napkins for a limited time at participating McDonald's.

Now when they're looking for it, it's very, very uncommon.

Some people apparently do get quite sick with it, but it's certainly very uncommon.

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Well, everybody knows in blood there's antibody, and they know that antibody can cause mediate protection.

Okay? That's pretty straightforward.

Now, the main antibody happens to be a protein which is given the term IgG, immunoglobulin G. It's now known that immunoglobulin G is made up of four different types, which are called subclasses.

Immunologists are incredibly imaginative.

Only immunologists, certainly mucosal immunologists, go to very boring places for meetings and call it IgG1, IgG2, IgG3 and IgG4.

IgG4 is present in most people in very small amounts.

Get your gear for the trail in store or at REI.com.

[laughs]

I, I think, I, I think what, to me, the, the central issue of what we got wrong is, a- and this has been happening, Chris, over a period of time.

Uh, we, we lost the regulatory influence of high quality medical people who have provided a brake on information and ensured that we traveled in a good direction.

Uh, and that had been going on for probably 20 or 30, 40 years.

And I, I think it's, we all live in a, a silo and, and my silo of course is health.

And, um, I've seen this very clearly and I think what COVID did was bring this out.

And, uh, with Tony Fauci, uh, not saying anything, uh, we- we've lost that, that opportunity to really, uh, get answers to questions.

And so, so this scary kind of thing, um, was coming over the population and behind it all, the pharmaceutical companies weren't prepared to put their backsides on the line in a legal backing of their vaccines.

[laughs]

I, I think, I, I think what, to me, the, the central issue of what we got wrong is, a- and this has been happening, Chris, over a period of time.

Uh, we, we lost the regulatory influence of high-quality medical people who have provided a brake on information and ensured that we traveled in a good direction.

Uh, and that had been going on for probably 20 or 30, 40 years.

And I, I think it's...

W- we all live in a, a silo, and, and my silo, of course, is health.

And, um, I've seen this very clearly, and I think what COVID did was bring this out.

And so, so this scary kind of thing, um, was coming over the population, and behind it all, the pharmaceutical companies weren't prepared to put their backsides on the line in a legal backing of their vaccines.

got to say that.

No, he really is.

John had come up with the idea that there is a commonality between the protection mechanisms of the different mucosal surfaces of the body, be it the lung, the gut, the eye.

And when I arrived, he was on the verge of proving this by showing with transfer of cells in rabbits from payous patches as a major factory of cells to various mucosal tissues, particularly in the lung.

And so I got particularly interested in that communication between the payous patch, which are little factories of cells in the small bowel, the lining of the small bowel.

And we... developed these ideas to include T lymphocytes as well as B lymphocytes so all the different armamentaria of the so-called adaptive or specific immune response were generated and the interesting thing is they weren't generated in the lungs they were generated in the post patch by the antigens Be they virus, bacteria being swallowed into the gut.

As we sit here, we sit here for 24 hours.

than every day.

just it's just i just find this isn't this amazing i mean what sort of innate mechanisms are there that keep us sterile just that just those few millimeters under the surface of the skin

Well, I think that's a really astute observation, John.

Two things need, I think, to be said.

The first is that you're absolutely right.

The body, for all intents and purposes, is sterile and needs to stay that way.

The finely balanced mechanisms, the metabolism...

The way in which the organs and cells talk to each other, the tissues behave, requires no bacteria, no viruses.

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in the world.

And we just don't have enough cells to make hundreds and thousands of cells capable of marshaling an army of specificity to kill those bacteria or viruses.

So the body's very clever.

It, it, it adopted what had been around since the earthworm and very primitive organisms called the, uh, innate immune system.

And this is a system of protection that predates both our knowledge and also humans going way back to the, uh, invertebrate kingdom.

It predates that to coming up with a system that could protect against infection without having antibodies or specificity.

Go ahead, please

I think

it's really important, too, to understand this, Alex, because I'm an immunologist.

I'm very pro-vaccination.

What I'm not pro is vaccination using technology that leads to harm more often than it leads to good.

So, I mean, very briefly, the history of vaccination began when Louis Pasteur found that if he took a bacteria and kept growing and growing and growing it, it lost its mojo.

It lost its capacity to cause harm, but kept its capacity to stimulate immunity.

And so that was the attenuated type of vaccine.

But tell me about it.

But it's just been ignored.

No.

Well, Okay, well, very briefly, my view is that, as an immunologist and a clinical immunologist, that all the various different vaccines you want to use against an inhaled virus, which is likely to be the usual pandemics that we see, flu, COVID, et cetera, they attack a system of the immune system process that is different to the one you inject the vaccine into yeah and so the bottom line is that it's the biology of the process that's going to dominate the outcome not whether you use a messenger rna vaccine a dna vaccine or a squashed up virus or whatever it's the limitations are set by the biology of that interplay between the virus and the local immune response and the suppression of immunity that goes with that to prevent overactive inflammation in the airways, full stop.

So my group, we said initially, we didn't say this, but we said this through COVID when we realised what was happening, that you're never ever going to have a fantastic virus against a pandemic.

Very, I mean, it's got to be...

A fantastic vaccine, yeah.

Sorry, a fantastic vaccine against a pathogenic virus coming in like this.

Yeah.

We have another reason for doing this video today, don't we? What have we got in

mind? Well, John, the main reason was to give you and Chris a little break given the intensity of recent years.

But being here, the professionals in health and legal and pharmaceutics, et cetera, got together and said, why don't we use this as an opportunity to bring together The best of the best that we can find in Australia that are concerned that we may not be getting as much out of reviews of the COVID problem as we should.

And so in a sense, we're looking at doing the Royal Commission that the government probably didn't want to do.

And so on Tuesday, the 2nd of April, we're having a day, a symposium, where we have a terrific array of individuals, including yourself, John, where we're looking at different aspects, the way we've seen the COVID pandemic, how it's been handled, the good, the bad, and probably the indifferent, and particularly looking at ways we can go ahead.

Because We started COVID with a plan and the plan didn't sort of go to plan, it disappeared and a narrative took over.

And the question really is, was the narrative as good as the plan that we had in place? And this is going to be the Australian Medical Professionals Exactly.

I mean, what are the sort of standout issues and problems for you? What sort of things will we be talking about in your presentation, for example?

Mm

... three studies.

Uh, the first is that they did... looked at hospital patients who'd been very sick for quite a period of time.

Now, everyone viewing this video knows that if you have a viral infection, you treat it in the first two days.

Or if you have a herpes simplex involving, uh, cold sores, uh, or vaginal, uh, sexually, uh, transmitted, uh, herpes, you can treat it very effectively, uh, with an antiviral given in the first two, maybe three days.

Uh, if you get herpes zoster, shingles, everyone knows you go straight to the doctor and get treated, and it's effective.

What you don't do is wait a week, because it doesn't work.

Mm-mm.

Now we're on a rather unique trail, so what is this trail and where are we? We're

on the Charles Darwin Walk and we're on it today is the opening of this new Charles Darwin Walk and it's called Charles Darwin Walk because in 1836 when Darwin was doing his around the world trip with Fitzroy on the Beagle, they stopped in Sydney for just under three weeks and while the ship was being repaired and revitalized Darwin smartly decided after being open only for about 18 years he would cross the mountains and in those days there was a sort of halfway stop called Weatherboard which we now know as Wentworth Falls where we are today and And he was staying at Weatherboard and they said, look, we've got to follow this creek.

So I'm sure you can see, I hope you can see the creek.

We're on the walk that runs down beside the creek.

And at the end of that creek, there's this massive waterfall.

And it's a dramatic fall, which surprised and impressed Darwin and actually led to some of the ideas that he later incorporated in his theory of evolution.

A

bit of everything.

Mm.

Now, I think they're the best around.

What they've done is right from the f- the rollout of the vaccine in 20, early 2021, they've been following each vaccine and each booster that's come out and looked at the protection.

Now, if you look carefully, uh, at the, uh, the vertical, uh, the vertical, uh, um, numbers there-

Yes

...

the Cleveland Clinic, and there was a 27% increase in infections in those who were given the vaccine.

27% increase in number of infections in the vaccinated group.

the australian perspective one thing that immediately struck me robert was was was how consistent this is with the experience in the united kingdom um and and from what i can gather the experience in canada in the united states there seems to be uh international issues it's almost as if there's been some sort of international choreography here isn't it well

it's interesting you should say that i should say Right up front.

But this book came about a meeting that I met with Mel McCann, who is my co-editor here.

Mel is, she's not here, she's in northern Queensland.

But if she's watching, I'm a little embarrassed, but I'll have to say, Mel is one of the most remarkable family practitioners I've ever met.

Single-handedly, she initiated a class action at her cost.

largely at her cost for the vaccine damaged in Australia.

that had, as far as I understand it, a first-class pandemic plan in place for decades.

And

again, this goes against the fundamental axioms of health care.

I mean, you know, we trained up, we were involved in teaching nurse practitioners.

And we said something like 80% of what you get, the information comes from what your patient tells you.

You might do a bit

of a physical exam that gives you a bit of extra information and a few blood tests.

But, you know, basically you listen to your patient.

And, you know, the famous McCaffrey saying about pain, pain is what the patient says it is existing when they say it does.

How good are genetic vaccines?

Well, I think this is just an introductory slide, and it identifies the five issues that I wanted to talk about.

The first was, how good are genetic vaccines? The second was, how dangerous are genetic vaccines? And then the third, which I think is a very important one, is what is the most effective therapy for COVID? I specifically am interested in the post-COVID vaccination syndrome because, as you'll see from these slides, this is one of the best kept secrets.

I see it very much as the COVID legacy.

It is what society has been left as a result of our interventions.

I thought we should look at where we're going and what we haven't learned and the mistakes that we really need to address for the future.

Sounds interesting and very beneficial for individuals and for society as a whole.

Right, well, this is the first topic, and that is how good are genetic COVID vaccines? And the main points that I wanted to make with this slide was that any vaccine which is injected to prevent or modify the outcome of an inhaled pathogen, a virus or a nasty bacteria, has to fit in with the biology of the whole system.

Yeah,

it's coming from the E. coli or whatever bacteria they're using.

In the middle, it's just to remind us that we now know quite clearly that the old adage that certainly we're all taught at university is that the DNA information goes to RNA, goes to protein.

So you actually translate the genetic information, the coding, into the structure and function of proteins.

We now know quite clearly... that the messenger RNA, which is supposed to be between the DNA and the protein, as you can see on this graph, can actually reverse and put its message back into genomic DNA.

And that is scary.

It's been found recently in a number of cancers, the expression of spike protein, which can only come from a genetic base.

in fact be exactly

It's a stimulated autoimmunity, really, isn't it?

Yeah.

And that wonderful graph done by the Japanese showing the mortality peaking at three to four months, that really defines that wave.

Because within that wave, you're getting just recently, I think this week came out that individuals between 20 and...

Males between 20 to 40 had a highly significant sevenfold increase in heart attacks if they were vaccinated.

So all that's really occurring within that that frame.

And then there's the second frame, which begins at six months, because we say someone's got post-COVID vaccine syndrome or COVID.

this is really also two very important graphs.

Exactly.

80% of people with the athletes was pretty much 95% of the athletes had been exposed, had marker symptoms.

And this is the interesting thing, that the people who have the Epstar-Barr virus and who are prone to getting fatigue never quite make the right sort of T cell response.

And I'll give you two quick examples, and these relate exactly to the situation that I'm certainly seeing and has recently been published to some extent with long COVID.

If you look at antibody production, the antibodies are made by a cell given the very imaginative title of a B cell, a B lymphocyte.

But the B lymphocyte requires T lymphocytes to help them.

So we have these helper T cells.

So moving on to the sort of, um, management strategies that we're going to use, Robert, I mean, I mean, the question is of course, what, what the heck do we, do we do about this? So, um, do we need to promote a more effective immunity within the respiratory tract and how would we do that?