Collagen, type V, alpha 1

The next gene is PPAR-GC1A for mitochondrial biogenesis and endurance capacity.

So the ACE for cardiovascular response to training, the COL1A1 and COL5A1 for collagen repair and soft tissue injury risk.

Then there's the VDR and the inflammatory genes for recovery capacity.

And one that's really important warning here, because I watch people get this wrong constantly, a result that's saying you're built for endurance does not mean endurance is good for you it means that if you choose to compete you could be better than average at it that's a different statement altogether so performance and health are not the same thing and they frequently worked directly against each other so being an elite athlete is not evidence of being healthy that one well um i say that if someone has really done real damage to myself in the process of what i did from an athletic perspective um what the recovery genes actually give you is the ability to plan your recovery deliberately instead of hoping for the best i had a client last week we're doing genetics and yeah he's going to be better at more of the endurance based stuff and he's going to need recovery built in so not just doing back-to-back sessions he needs to have time off and that's part of his whole training plan if that makes sense If your collagen repair is slow and your inflammatory genes are loud, the answer is not don't exercise and you're fragile.

all the time in the world for you, which is really cool.

All right, our last question comes from Grayson, and they're asking, does a genetic mutation with the risk always mean that you have the condition, or does the mutation have to occur at a certain location on the gene, for example, CEDS? If someone has a mutation in the COL5A1 or 2 gene and symptoms, does that mean they have CEDS, or does that mutation have to occur in the specific location on the gene? i understood zero percent of that question so that's all you draw

okay so um so for ceds classical eds um we know that this is caused by specific pathogenic variants and call 5a1 or call 582.

Um, not every single mutation.