acute respiratory distress syndrome

The lung also forms colloid osmotic gradients that help remove transidated fluids.

But those protections are easier to overwhelm when there's pulmonary inflammation, glycocalyx injury, sepsis, ARDS, trauma, or contusion.

Acute respiratory distress syndrome, or ARDS, is a form of inflammatory lung injury characterized by increased pulmonary capillary permeability, bilateral lung infiltrates, and respiratory dysfunction that can't be explained by left-sided heart failure or simple fluid overload.

Because the barrier is disrupted, fluid leaking into the alveoli is typically protein-rich.

The 2021 study titled Fluid Therapy in Pulmonary Disease, How Careful Do We Need to Be? highlights an important species difference.

I was admitted.

They diagnosed it as ARDS, which was acute respiratory distress syndrome, also known as lung failure.

And being a syndrome, it did not react to antibiotics.

So they said, you know, you've got a 38% chance of making it.

Gastric contents enter the lungs and the chemical injury produces inflammation.

That can result in hypoxemia, bronchospasm, decreased lung compliance, pulmonary edema, and in severe cases, an ARDS-like picture.

Aspiration pneumonia is different.

That's an infectious process caused by aspiration of bacteria containing material.

It turns out with Hantavirus and Ebola virus, the first four pathways, NF-kappa B, NLRP3 inflammasomes, NRF2, and prostaglandins are all relevant to Hantavirus and Ebola virus.

And Dapsone showed in the first wave of the COVID, when people were dying from ARDS, acute respiratory distress syndrome, when there was a whiteout of their lungs because the lungs have 140 times more glutathione than any place else in the body so if you get an infection in your lung from hantavirus or ebola virus and you've used up your glutathione because you're dealing with infections and toxins you have nothing to protect your alveoli and your bronchi from all the inflammation you get ards they showed with dapsone it was a small study with covid but the 40 patients who took dapsone not one of them died from ards so when i looked at hantavirus and ebola virus dapsone blocks myeloperoxidase, and dapsone blocks NLRP3 inflammasomes, which are relevant to these viral infections.

So NLRP3 inflammasomes is a pathway that is seen in Alzheimer's.

So the study that was done in Korea with these 4,000 patients over 15 years, They took 100 milligrams of dapsone, not the doses I'm using for Lyme.

So it's like, maybe I have an answer, maybe I don't.

But this is what science says is at least the next logical thing to protect yourself.

And based on what I've looked at, I think based on the prior studies in COVID-19 where Dapsone stopped ARDS, even though it's a small study, that is the main reason why people die from hantavirus.

I mean, it's a little bit different with Ebola, which also really affects the cells and destroys it.

Here's an article of scientific interest from the UB News Center out of Buffalo.

Lung inflammation and injury can quickly turn into acute respiratory distress syndrome, that's ARDS, the abbreviation, in patients with pneumonia, COVID-19, and other conditions.

ARDS, that's the acronym, affects more than 200,000 people annually in the United States.

Of these, 30 to 50 percent die, according to Zhenya Wang, Ph.D., SUNY Empire Innovation Professor in the School of Pharmacy and Pharmaceutical Sciences at the University of Rochester.

I'm sorry, at the University at Buffalo.

Wang recently received a $2 million four-year grant from the National Heart, Lung and Blood Institute, the NHLBI, to look for a safer and more effective treatment using omega-3 fatty acids.

He is collaborating with Elsa Bo Ghanem, Ph.D., associate professor in the Department of Microbiology and Immunology at the Jacob School of Medicine and Biomedical Sciences at UB.

Acute lung inflammation injury is a dysregulated inflammation disease, and it quickly precipitates to the more severe ARDS, Wang said.